Michael Fischbein

Back to Profile

Publication Details

  • Interleukin-16 deficiency suppresses the development of chronic rejection in murine cardiac transplantation model.

    Kimura N, Itoh S, Nakae S, Axtell RC, Velotta JB, Bos EJ, Merk DR, Gong Y, Okamura H, Nagamine CM, Adachi H, Kornfeld H, Robbins RC, Fischbein MP. J Heart Lung Transplant. 2011; 30 (12): 1409-17

    IL-16 promotes the recruitment of various cells expressing CD4, a receptor for IL-16. The precise role of IL-16 in transplant rejection remains unknown; therefore, the present study investigated the contribution of IL-16 to the development of chronic rejection in heart transplants.

    PubMedID: 22055099

Stanford Medicine Resources:

Footer Links: